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Survodutide 10mg
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Buy Survodutide 10mg

GLP-1/glucagon dual agonist for obesity and NASH with superior fat liver targeting

Cheap Survodutide 10mg — Survodutide (BI 456906) is a Phase III GLP-1/glucagon receptor dual agonist from Boehringer Ingelheim with particular efficacy against non-alcoholic steatohepatitis (NASH) and obesity.

  • GLP-1/glucagon dual agonism for obesity and NASH simultaneously
  • 14.9% body weight loss in Phase II at optimal doses
  • Strongest liver fat reduction of any dual agonist in development
  • Phase III NASH trial — potential first NASH approval
  • Reduces liver steatosis, inflammation, and fibrosis
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Survodutide: The Phase III GLP-1/Glucagon Dual Agonist Targeting Obesity and NASH Simultaneously

NASH Disease Modification

Survodutide is in active Phase III trials as a potential first approved NASH treatment — targeting liver fat, inflammation, and fibrosis simultaneously through glucagon receptor hepatic activation.

Hepatic Glucagon Action

Glucagon receptor activation drives hepatic fatty acid oxidation and reduces de novo lipogenesis — directly targeting the liver fat accumulation at the core of NASH pathology.

14.9% Bodyweight Loss

Phase II obesity data showing 14.9% weight reduction at 46 weeks — competitive with the best established GLP-1 agonists and without the cardiovascular outcome data limitations.

Obesity-NASH Continuum

A single compound addressing both obesity and its most dangerous metabolic complication — survodutide is the only agent in late-stage development designed to treat this disease continuum simultaneously.

The Science Behind Survodutide 10mg

Next-Generation GLP-1/Glucagon Dual Agonism

Survodutide (BI 456906) is a once-weekly subcutaneous dual GLP-1/glucagon receptor agonist developed by Boehringer Ingelheim, currently in Phase III trials for obesity and NASH. Its glucagon receptor component is particularly potent for hepatic fat reduction — making Survodutide the dual agonist with the strongest evidence for treating both obesity and the associated liver disease (NASH) that is increasingly prevalent in obese populations.

NASH Targeting: The Glucagon Advantage

Glucagon receptor activation in hepatocytes directly stimulates hepatic fatty acid oxidation and reduces de novo lipogenesis — the two key drivers of non-alcoholic fatty liver disease. Phase II NASH trials with Survodutide showed marked improvements in liver steatosis, inflammation, and fibrosis scores, positioning it as a potential disease-modifying treatment for NASH — a condition with no approved pharmacotherapy.

Phase III Pipeline and Clinical Data

Phase II obesity trials showed 14.9% body weight loss over 46 weeks at optimal doses — competitive with semaglutide and other approved GLP-1 agonists. The Phase III NASH trial is a landmark study with potential to establish Survodutide as the first approved treatment for this condition, which affects over 1.5% of the global population and has no current pharmacological standard of care.

Complete Survodutide 10mg Benefits

  • GLP-1/glucagon dual agonism for obesity and NASH simultaneously
  • 14.9% body weight loss in Phase II at optimal doses
  • Strongest liver fat reduction of any dual agonist in development
  • Phase III NASH trial — potential first NASH approval
  • Reduces liver steatosis, inflammation, and fibrosis
  • Once-weekly dosing for sustained weight and liver fat control
  • Addresses the obesity-NASH continuum with a single agent

Survodutide 10mg Dosing Protocol

Reconstitution: Add 2–3mL bacteriostatic water.

Dosing:

Phase II protocol: 1.2–4.8mg subcutaneous weekly (dose escalation)

Start: 0.3mg weekly, escalate monthly to target

Maximum studied: 4.8mg weekly

Once-weekly subcutaneous injection

All information on this site is for educational purposes only. Always consult with a qualified healthcare provider before use. COA documentation is available for all products.

Survodutide 10mg

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$159.99
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